Joint Action of Trichoderma hamatum and Difenoconazole on Growth of a Phytopathogen Sclerotinia sclerotiorum under Laboratory Conditions
Dan Yü, Chuchu Li, Yü Huang and Zhen Huang*
College of Agriculture, South China Agricultural University, Guangzhou 510642, P.R. China
Fig. 1.
Inhibition of Trichderma hamatum on mycelial growth and Sclerotia formation of Sclerotinia sclerotiorum.
Fig. 2.
The inhibition of EC50 of T. hamatum cell-free culture supernatant on mycelial growth of S. sclerotiorum.
Fig. 3.
The inhibition of T. hamatum cell-free culture supernatant on mycelial growth inhibition of S. sclerotiorum after treated in 70°C for 10 min. Data on mean (±SE) inhibitory percentage were subjected to arcsine transformation prior to computation
Fig. 4.
The inhibition of T. hamatum cell-free culture supernatant on mycelial growth inhibition of S. sclerotiorum after treated in 70°C for 0, 10, 20 and 30 min. Data on mean (±SE) inhibitory percentage were subjected to arcsine transformation prior to computation. Means±SE in time dots (the column) marked with different letters are significantly different (Tukey’s HSD, a=0.05).
Fig. 5.
SDS-PAGE of protein from T. hamatum cell-free culture supernatant after treated in 70°C for 0, 20 and 30 min.
Fig. 6.
The inhibition of T. hamatum ethylacetate on mycelial growth of S. sclerotiorum. Data on mean (±SE) inhibitory percentage were subjected to arcsine transformation prior to computation.
Fig. 7.
The inhibition of T. hamatum ethylacetate extracts on sclerotia production of S. sclerotiorum. Data on mean (±SE) inhibitory percentage were subjected to arcsine transformation prior to computation.
Fig. 8.
The inhibition of difenoconazole on mycelial growth of S. sclerotiorum. Data on mean (±SE) inhibitory percentage were subjected to arcsine transformation prior to computation.